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Comment on:
Psychiatric effects of ephedra: addiction.
Miller SC.
Publication Types:
PMID: 16263877 [PubMed - indexed for MEDLINE]
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Tardive dystonia associated with ziprasidone.
Papapetropoulos S, Wheeler S, Singer C.
Publication Types:
PMID: 16263868 [PubMed - indexed for MEDLINE]
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Secondary mania in older adults.
Brooks JO 3rd, Hoblyn JC.
Department of Psychiatry and Behavioral Sciences, Stanford University School of Medicine, Stanford, CA 94305, USA. johnbrooks@stanford.edu
Publication Types:
PMID: 16263839 [PubMed - indexed for MEDLINE]
Hepatotoxicity associated with supplements containing Chinese green tea (Camellia sinensis).
Bonkovsky HL.
Publication Types:
PMID: 16389263 [PubMed - indexed for MEDLINE]
Hashimoto encephalopathy with pegylated interferon alfa-2b and ribavirin.
Deutsch M, Koskinas J, Tzannos K, Vassilopoulos D, Mailis A, Tolis G, Hadziyannis S.
Academic Department of Internal Medicine, Hippocration General Hospital Athens, Greece. kostam@ath.forthnet.gr
OBJECTIVE: To report an instance of Hashimoto encephalopathy probably resulting from pegylated interferon alfa-2b and ribavirin. CASE SUMMARY: A 36-year-old woman with a 10-year history of autoimmune thyroiditis presented with symptoms and signs consistent with Hashimoto encephalopathy during therapy with pegylated interferon alfa-2b and ribavirin for chronic hepatitis C. DISCUSSION: Hashimoto encephalopathy is a rare autoimmune condition that occurs in patients with Hashimoto thyroiditis and high titers of antithyroid antibodies. It is characterized by a variety of nonspecific neuropsychiatric symptoms, increased cerebrospinal fluid protein level, and abnormal brain imaging and electroencephalogram. Prompt response to corticosteroids is observed in most cases. As of August 29, 2005, this is the first report of such an association. An objective causality assessment revealed that the Hashimoto encephalopathy was probably caused by the patient's medications. CONCLUSIONS: Hashimoto encephalopathy may rarely be triggered by interferon alfa therapy in susceptible patients.
Publication Types:
PMID: 16159996 [PubMed - indexed for MEDLINE]
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Rhabdomyolysis after correction of hyponatremia in psychogenic polydipsia possibly complicated by ziprasidone.
Zaidi AN.
Department of Internal Medicine, The Pennsylvania State University, The Penn State Milton S Hershey Medical Center, Hershey, PA 17033-2360, USA. azaidi@psu.edu
OBJECTIVE: To report a case of rhabdomyolysis related to correction of hyponatremia secondary to psychogenic polydipsia, possibly complicated by the use of ziprasidone. CASE SUMMARY: A 50-year-old white man treated for 3 weeks with ziprasidone 40 mg twice daily for chronic paranoid schizophrenia was admitted to the intensive care unit after a witnessed generalized seizure. Marked hypotonic hyponatremia was present secondary to psychogenic polydipsia. After correction of hyponatremia with intravenous NaCl 0.9%, he developed a substantial elevation in the creatine kinase level without any evidence of muscle trauma, stiffness, or swelling or any signs of neuroleptic malignant syndrome. Renal failure or compartment syndrome did not complicate the clinical picture. DISCUSSION: It is well known that severe hyponatremia can cause neurologic complications such as stupor, seizures, and even coma. Hyponatremia from water intoxication (n = 28) and its correction with intravenous fluids (n = 2) may cause non-neurologic complications such as rhabdomyolysis. An explanation may lie within the calcium-sodium exchange mechanism across the skeletal myocyte or the failure of cell volume regulation secondary to extracellular hypo-osmolality. Neuroleptic medications have been linked to the development of rhabdomyolysis, with antipsychotics being the primary offenders. As of August 2005, there has been only one reported case of rhabdomyolysis related to correction of hyponatremia complicated by an atypical antipsychotic (clozapine). It is possible that ziprasidone, like clozapine, may enhance muscle cell permeability leading to rhabdomyolysis under similar conditions. CONCLUSIONS: Psychiatric patients treated with atypical antipsychotic medications should be closely monitored for rhabdomyolysis during correction of hyponatremia, thus permitting prompt therapy to limit its complications.
Publication Types:
PMID: 16131536 [PubMed - indexed for MEDLINE]
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Antidepressant-induced adverse reactions in a patient with hemorrhagic stroke.
Bogdanovic Z, Nalamati JR, Kilcullen JK, Dhuper S.
Department of Medicine, North Central Bronx Hospital, Bronx, NY 10467-2410, USA.
OBJECTIVE: To report a case of antidepressant-induced adverse drug reactions in a patient with hemorrhagic stroke. CASE SUMMARY: A 56-year-old man developed life-threatening adverse reactions after fluoxetine was added to his previously prescribed regimen of buspirone and olanzapine. One week after starting fluoxetine 60 mg/day, the patient developed syndrome of inappropriate antidiuretic hormone secretion and serotonin syndrome concurrently. The patient had experienced a hemorrhagic stroke before the adverse drug reactions occurred. DISCUSSION: A patient with a history of hemorrhagic stroke developed serious adverse drug reactions when fluoxetine was added to his drug therapy. When the combination therapy was stopped, all adverse effects gradually disappeared and laboratory abnormalities were corrected. The likelihood that the adverse reactions were caused by fluoxetine is probable according to the Naranjo probability scale. In addition, a history of stroke may be a risk factor for the development of such reactions. CONCLUSIONS: Today, patients with depression after experiencing a stroke are treated more effectively, but antidepressant-induced adverse drug reactions may be serious. A growing number of patients are treated for post-stroke depression; they require close supervision and careful dosing of antidepressants to prevent full-blown adverse reactions from occurring.
Publication Types:
PMID: 16091417 [PubMed - indexed for MEDLINE]
Successful treatment with oral isotretinoin of acneiform skin lesions associated with cetuximab therapy.
Gutzmer R, Werfel T, Mao R, Kapp A, Elsner J.
Publication Types:
PMID: 16181478 [PubMed - indexed for MEDLINE]
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Dermatitis during efalizumab treatment in a patient with psoriasis vulgaris.
de Groot M, de Rie MA, Bos JD.
Publication Types:
PMID: 16181475 [PubMed - indexed for MEDLINE]
A fatal poisoning with 5-methoxy-N,N-diisopropyltryptamine, Foxy.
Tanaka E, Kamata T, Katagi M, Tsuchihashi H, Honda K.
Department of Legal Medicine, University of Tsukuba, Ibaraki 305-8575, Japan.
A fatal overdose involving case by 5-methoxy-N,N-diisopropyltryptamine (5-MeO-DIPT) is reported. 5-MeO-DIPT and its two metabolites, 5-hydroxy-N,N-diisopropyltryptamine (5-OH-DIPT) and 5-methoxy-N-isopropyltryptamine (5-MeO-NIPT), were identified by LC-MS. The level of 5-MeO-DIPT, 5-OH-DIPT and 5-MeO-NIPT in blood and urine was 0.412, 0.327 and 0.020mug/ml, and 1.67, 27.0 and 0.32mug/ml, respectively. These blood and urine levels were higher than published data for such poisoning.
PMID: 16406422 [PubMed - as supplied by publisher]
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